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I have been using Iressa for about two month to treat cancer in my lungs. My doctor says this medication prevents cancer cells from growing. Of course, it was only my impression, but I thing Iressa improved my condition because I started feeling myself much better. And after using the medication for about a month my doctor carried out a number of tests and stated that the amount of cancer cells I my lugs decreased. I was really happy to hear that! Not I finally have hope thanks to Iressa!



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I was diagosed with inoperable NSCLC in March 11. I am taking Iressa as a first line treatment. I had no side effects for 2 weeks then the rash on my face started. I am female so was able to cover this with makeup. My skin became very dry too but moisturizers helped. I had tummy cramps which started at the same time, no diarrhea initially as I was still taking morphine from unsuccessful surgery. After 6 weeks I had my first scan. My tumor that had been 11cms was now 8.5cms, this was a pleasant surprise as I was told the Iressa would not shrink it but just stop it from growing. Since stopping the morphine I now suffer from tummy cramps and diarrhea most days, usually at night. I feel it is a small price to pay. I take Imodium which helps.

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Without therapy, most patients survive 4-6 weeks, with death occurring because of progressive neurologic dysfunction.

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Finding the tumor copycat: Therapy fails, patients don't.

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Afatinib reduced the size of target lesions in 79% of all patients during the treatment period (), with nine patients (16%) having at least a 30% reduction in tumor size.

Not all TKIs exert the same toxicity on the heart muscle, indicating that this is not a class toxic effect.

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Mutations in p53, a tumor suppressor gene, were among the first genetic alterations identified in astrocytic brain tumors.

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Results: The combination of metformin with gefitinib induced a strong antiproliferative and proapoptotic effect in NSCLC cell lines that harbored wild-type LKB1 gene.

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Schwaegerle SM, Bergfeld WF, Senitzer D: Pyoderma gangrenosum: a review.

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The Iressa Pan-Asia Study (IPASS) was the first phase III randomized trial that demonstrated superior outcome with first-line EGFR-TKI treatment in patients with EGFR-mutant NSCLC when compared with platinum-based chemotherapy in a retrospective subgroup analysis ().

Treatment goals include improvement or stabilization of the patient's neurologic status, prolongation of survival, and palliation.

The most common adverse events in the gefitinib group were rash and elevated levels of aspartate aminotransferase or alanine aminotransferase, and in the chemotherapy group, appetite loss, neutropenia, anemia, and sensory neuropathy ( Table 3 Common Toxic Effects in the Safety Population, According to Treatment Group.

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With gefitinib, longer median survival was seen in patients with a high versus low EGFR-gene-copy number (8.

Results are anticipated and should help determine if neratinib is active or inactive in this select patient population that is enriched for patients with acquired resistance to TKIs because of T790M or MET amplification in the EGFR-mutated arm.

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The WJTOG0203 () and INFORM () trials used gefitinib whereas SATURN () and IFCT-GFPC 0502 () showed similar benefits for erlotinib.

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After 3 cycles of chemotherapy, her disease progressed in both lungs and bones.

También se agregó que de manera similar al análisis de la SSA, la SG se estratificó según el tipo de mutación en EGFR y el origen étnico.

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Gefitinib treatment in patients with postoperative recurrent non-small-cell lung cancer harboring epidermal growth factor receptor gene mutations.

Phase I single-dose study of intracavitary-administered iodine-131-TM-601 in adults with recurrent high-grade glioma.

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Our results should motivate the development of alternative EGFR inhibitors or inhibitors of downstream targets of EGFR such as phosphatidylinositol 3'-kinase or STAT5 for patients with EGFR-mutant tumors with acquired resistance to anilinoquinazoline inhibitors.

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EGFR) is highly responsive to EGFR tyrosine kinase inhibitors such as gefitinib, but little is known about how its efficacy and safety profile compares with that of standard chemotherapy.

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We performed co-immunoprecipitation and Western blot assays in V1801-treated NCI-H1975 cells, and found that V1801 but not gefitinib enhanced Mcl-1-Noxa binding affinity ().

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In the Iressa in NSCLC Versus Vinorelbine Investigation in the Elderly (INVITE) trial, 21 untreated elderly patients (aged 70 years or older) were randomized to gefitinib vs vinorelbine, and the treatments were found to be statistically similar, with a response rate of only 3% for gefitinib.

Nivolumab is a fully human monoclonal antibody that inhibits the programmed death-1 (PD-1) co-inhibitory immune checkpoint expressed on tumor cells and infiltrating immune cells.

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Proliferating cell nuclear antigen (PCNA) was detected using a PCNA Staining kit (Zymed Laboratories).

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Furthermore, real-time quantitative PCR analysis showed that gefitinib reduced FOXM1 mRNA transcripts in the sensitive SKBR3 and BT474 but not in the resistant MDA-MB-453 cell lines ().

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ASCO guidelines recommend using white cell stimulating factors when the risk of febrile neutropenia, secondary to a recommended chemotherapy regimen, is approximately 20 percent and equally effective treatment programs that do not require white cell stimulating factors are unavailable.

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Small-cell Lung Cancer: Quality of Life Results From a Randomized Phase III Trial.

A) Whole-cell lysates were analyzed by immunoblotting using antibodies respectively specific against cleaved caspase-3 and β-actin.

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EGFR testing of tumors at diagnosis from patients presenting with stage I, II, or III disease is encouraged but the decision to do so should be made locally by each laboratory, in collaboration with its oncology team.

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